SUMMARY
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Equinovarus Foot is an acquired foot deformity commonly seen in pediatric patients with cerebral palsy, spina bifida, and Duchenne Muscular Dystrophy that present with a equinovarus foot deformity.
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Diagnosis is made clinically with presence of an inverted heel with a supinated forefoot, often associated with pain and callous formation along the lateral border of the foot.
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Treatment ranges from bracing to tendon transfers to osteotomies depending on the underlying etiology, severity of deformity, and rigidity of contracture.
EPIDEMIOLOGY
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Incidence
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common foot deformity seen with
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cerebral palsy (usually spastic hemiplegia)
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Duchenne muscular dystrophy
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residual clubfoot deformity
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spina bifida
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tibial deficiency (hemimelia)
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though this condition is very rare
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ETIOLOGY
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Pathophysiology
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pathomechanics
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imabalance of invertors and evertors (invertors overpower the evertors)
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relative overpull of
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tibialis posterior and/or
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tibialis anterior
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gastoc-soleus complex
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example: in cerebral palsy
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the causative muscles for the varus are the
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anterior tibialis (AT) in 1/3 of patients
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posterior tibialis (PT) in 1/3 and
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both the AT and PT in the remaining 1/3
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foot deformity muscle imbalance overview
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PRESENTATION
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Symptoms
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pain
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painful weight bearing over the lateral border of the foot
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instability
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during stance phase
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results in shortened single limb stance
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poor shoe and/or brace fitting and shoe wear problems
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Physical Exam
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inspection
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inverted heel (tibialis posterior typically implicated)
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supinated forefoot (tibialis anterior)
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callous and pain along lateral border
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intoeing gait (foot progression angle is more internal than knee progression angle)
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provocative tests
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active dorsiflexion of foot
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if foot supinates with dorsiflexion, the anterior tibialis is implicated
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confusion test
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indications
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used in those with poor selective motor control, as in CP, and cannot dorsiflex foot when asked)
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method
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patient performs active hip flexion (with or without resistance) while seated
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results in ankle dorsiflexion due to mass action pattern of leg
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if the foot supinates with dorsiflexion, the tibialis anterior is likely a contributing to the varus deformity
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Coleman block test
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indications
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to test rigidity of the varus deformity
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do not do this in children with limited balance such as CP
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method
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patient stands on a block with the first ray off the block
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if the varus corrects, the deformity is flexible
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manual manipulation of the hindfoot
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can be used to asses rigidity of the varus deformity
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passive eversion of the hindfoot past neutral demonstrates that the varus deformity is flexible
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IMAGING
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Radiographs
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recommended views
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AP + lateral of foot
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findings
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forefoot adduction is seen on the AP radiograph
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the talus and calcaneus are more parallel than in typical feet
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one can often "look down" the sinus tarsi through a visual hole there
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the calcaneus looks foreshortened on the lateral view
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the metatarsals are often "stacked" on the lateral view (instead of being in line with one another)
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stress fractures along the fourth and/or fifth metatarsal bases can develop secondary to repetitive load along the lateral border of the foot.
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STUDIES
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Dynamic EMG
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may be useful in distinguishing whether tibialis anterior and/or tibialis posterior is/are causing the varus in CP
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TREATMENT
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Nonoperative
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ankle foot orthosis (AFO)
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helps provide stability for the foot and a more stable base of support during gait
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should have a "wrap around" hindfoot component of the brace to help control the varus and minimize pressure points
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serial casting
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indication
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rigid deformity
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botulinum toxin injection into tibialis posterior and/or gastrocnemius
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indication
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flexible or dynamic deformities
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desire to delay surgery
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Operative
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gastrocnemius recession or tendoachilles lengtheing (TAL) for equinus
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indications
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fixed equinus unresponsive to non-operative measures
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gastrocnemius recession should be performed if the anke can be brought to neutral or above neutral with the knee flexed and hindfoot inverted, but not when the knee is extended
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TAL should be performed if the ankle can not be dorsiflexed to neutral with the knee flexed or extended
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split-posterior tibialis tendon transfer [SPOTT] or posterior tibial tendon lengthening (PTTL)
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indications
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soft tissue balancing is required if varus is flexible or rigid
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varus foot recalcitrant to non-operative measures and posterior tibialis contributing to varus (dynamic EMG, when available is helpful)
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tibialis posterior spastic in both stance and swing phase (continous activity)
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common patient: spastic hemiplegia in ages 5 to 7 years old
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technique
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SPOTT
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reroute half of tendon laterally and insert into peroneus brevis
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PTTL
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fractional lengthening of the tendon in the distal third of the lower leg
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either PTTL or SPOTT may be combined with SPLATT
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outcomes
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results for both surgeries are good, without clear indications for transfer versus lengthening
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split-anterior tibialis tendon transfer [SPLATT]
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indications
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overactive anterior tibialis on EMG
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when anterior tibialis contributes to varus foot, whether flexible or rigid varus deformity
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technique
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split anterior tibialis transfer to cuboid, peroneus tertius, or peroneus brevis
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may be combined with SPOTT or PTTL
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calcaneal osteotomy
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indications
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required for a rigid hindfoot varus deformity
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technique
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lateral closing wedge osteotomy (Dwyer) to incur valgus to the heel, OR
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lateral calcaneal sliding osteotomy to correct the varus
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typically combined with soft tissue balancing (as above)
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COMPLICATION
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Overcorrection (resultant valgus deformity)
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increased risk in
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children who undergo surgery at younger age
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children with diplegia (as oppose to hemiplegia)
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Wound complications
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most common with calcaneal osteotomy lateral incision
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risk decreased by using absorbable suture
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Hardware Pressure sores/ulcers
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from buttons on bottom of foot (from SPLATT to cuboid)
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has led some surgeons to always transfer SPLATT to peroneus tertius or brevis
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