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SUMMARY
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Knee dislocations are high energy traumatic injuries characterized by a high rate of neurovascular injury.
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Diagnosis is made clinically with careful assessment of limb neurovascular status. Radiographs should be obtained to document reduction.
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Treatment is generally emergent reduction and stabilization with assessment of limb perfusion followed by delayed ligamentous reconstruction.
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EPIDEMIOLOGY
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Incidence
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rare
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0.02% of orthopedic injuries
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likely underreported as approximately 50% self-reduce and are misdiagnosed
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Demographics
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4:1 male to female ratio
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Location
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tibiofemoral articulation (knee joint)
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Risk factors
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morbid obesity is a risk factor for "ultra-low energy" knee dislocations with activities of daily living
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PATHOPHYSIOLOGY
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Mechanism of injury
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high-energy vs low energy
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high energy is usually from MVC, crush injury, fall from a height, or dashboard injury resulting in axial load to a flexed knee
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low energy may be from an athletic injury or routine walking
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hyperextension injury leads to anterior dislocations
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posteriorly directed force across the proximal tibia (dashboard injuries) leads to posterior dislocations
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Associated injuries
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vascular injury
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nerve injury
- usually common peroneal nerve injury (25% incidence)
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tibial nerve injury is less common
- usually common peroneal nerve injury (25% incidence)
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fractures
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present in 60% of dislocations
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soft tissue injuries
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patellar tendon rupture
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periarticular avulsion
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displaced menisci
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ANATOMY
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Osteology
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the knee is a ginglymoid joint and consists of tibiofemoral, patellofemoral and tibiofibular articulations
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Ligaments
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PCL, ACL, LCL, MCL, and PLC are all at risk for injury
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main stabilizers of the knee given the limited stability afforded by the bony articulations
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Blood supply
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popliteal artery injuries occur often due to tethering at the popliteal fossa
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proximal - fibrous tunnel at the adductor hiatus
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distal - fibrous tunnel at soleus muscle
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geniculate arteries may provide collateral flow and palpable pulses masking a limb-threatening vascular injury
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Biomechanics
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the normal range of motion of 0-140 degrees with 8-12 degrees of rotation during flexion/extension
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CLASSIFICATION
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Descriptive
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Kennedy classification based on the direction of displacement of the tibia
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- Kennedy classification
(based on the direction of displacement of the tibia)
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Anterior (30-50%)
- most common
due to hyperextension injury
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usually involves tear of PCL
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an arterial injury is generally an intimal tear due to traction
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the highest rate of peroneal nerve injury
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Posterior (30-40%)
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2nd most common
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due to axial load to the flexed knee (dashboard injury)
- the highest rate of vascular injury based on Kennedy classification
has highest incidence of a complete tear of the popliteal artery
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Lateral (13%)
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due to a varus or valgus force
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usually involves tears of both ACL and PCL
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Medial (3%)
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varus or valgus force
- usually disrupted PLC and PCL
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Rotational (4%)
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usually irreducible
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posterolateral is most common rotational dislocation
- buttonholing of femoral condyle through the capsule
- Kennedy classification
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Schenck Classification
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based on a pattern of multiligamentous injury of knee dislocation (KD)
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- Schenck Classification
(based on the number of ruptured ligaments)
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KD I
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Multiligamentous injury with the involvement of the ACL or PCL
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KD II
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Injury to ACL and PCL only (2 ligaments)
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KD III
- Injury to ACL, PCL, and PMC or PLC (3 ligaments).
KDIIIM (ACL, PCL, MCL) and KDIIIL (ACL, PCL, PLC, LCL).
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KD IV
- Injury to ACL, PCL, PMC, and PLC (4 ligaments)
Has the highest rate of vascular injury (5-15%%)
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KD V
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Multiligamentous injury with periarticular fracture
- Schenck Classification
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PRESENTATION
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Symptoms
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history of trauma and deformity of the knee
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knee pain & instability
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Physical exam
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appearance
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no obvious deformity
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50% spontaneously reduce before arrival to ED
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may present with subtle signs of trauma (swelling, effusion, abrasions, ecchymosis)
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obvious deformity
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reduce immediately, especially if absent pulses
- "dimple sign" - buttonholing of medial femoral condyle through the medial capsule
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indicative of an irreducible posterolateral dislocation
- a contraindication to closed reduction due to risks of skin necrosis
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vascular exam
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priority is to rule out vascular injury on exam both before and after reduction
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serial examinations are mandatory
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palpate the dorsalis pedis and posterior tibial pulses on injured and contralateral side
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if pulses are present and normal
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does not indicate the absence of arterial injury
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collateral circulation can mask a complete popliteal artery occlusion
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- measure Ankle-Brachial Index (ABI) on all patients with suspected KD
- if ABI >0.9
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then monitor with serial examination (100% Negative Predictive Value)
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if ABI <0.9
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perform an arterial duplex ultrasound or CT angiography
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if arterial injury confirmed then consult vascular surgery
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- if ABI >0.9
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If pulses are absent or diminished
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confirm that the knee joint is reduced or perform immediate reduction and reassessment
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immediate surgical exploration if pulses are still absent following reduction
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ischemia time >8 hours has amputation rates as high as 86%
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imaging contraindicated if it will delay surgical revascularization
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if pulses present after reduction then measure ABI then consider observation vs. angiography
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neurologic exam
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assess sensory and motor function of peroneal and tibial nerve as nerve deficits often occur concomitantly with vascular injuries
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stability
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diagnosis based on instability on physical exam (radiographs and gross appearance may be normal)
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may see recurvatum when held in extension
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assess ACL, PCL, MCL, LCL, and PLC
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IMAGING
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Radiographs
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recommended views
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pre-reduction AP and lateral of the knee
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may be normal if spontaneous reduction
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look for asymmetric or irregular joint space
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look for avulsion fxs (Segond sign - lateral tibial condyle avulsion fx)
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osteochondral defects
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post reduction AP and lateral of the knee
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optional views
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45-degree oblique if fracture suspected
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CT
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indications
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fracture identified on post reduction plain films
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obtain post reduction CT for characterization of fracture
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findings
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tibial eminence, tibial tubercle, and tibial plateau fractures may be seen
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MRI
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indications
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obtain MRI after acute reduction but prior to hardware placement
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required to evaluate soft tissue injury (ligaments, meniscus) and for surgical planning
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TREATMENT
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Nonoperative
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emergent closed reduction followed by vascular assessment/consult
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indications
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considered an orthopedic emergency
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vascular consult indicated if
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pulses are absent or diminished following reduction
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if arterial injury confirmed by arterial duplex ultrasound or CT angiography
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immobilization as definitive management
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indications (rare)
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successful closed reduction without vacular compromise
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most cases require some form of surgical stabilization following reduction
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outcomes
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worse outcomes are seen with nonoperative management
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prolonged immobilization will lead to loss of ROM with persistent instability
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Operative
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open reduction
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indications
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irreducible knee
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posterolateral dislocation
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open fracture-dislocation
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obesity (may be difficult to obtain closed)
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vascular injury
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external fixation
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indications
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vascular repair (takes precedence)
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open fracture-dislocation
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compartment syndrome
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obese (if difficult to maintain reduction)
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polytrauma patient
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delayed ligamentous reconstruction/repair
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indications
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instability will require some kind of ligamentous repair or fixation
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patients can be placed in a knee immobilizer until treated operatively
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improved outcomes with early treatment (within 3 weeks)
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TECHNIQUE
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Closed reduction
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approach
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anterior dislocation - traction and anterior translation of the femur
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posterior dislocation - traction, extension, and anterior translation of the tibia
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medial/lateral - traction and medial or lateral translation
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rotatory - axial limb traction and rotation in the opposite direction of deformity
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splinting
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20 to 30 degrees of flexion
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Open reduction
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approach
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midline incision with a medial parapatellar arthrotomy
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soft tissue
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the medial capsule may need to be pulled over medial condyle if buttonholed
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acute associated soft tissue injuries (patellar tendon rupture, periarticular avulsion, or displaced menisci) may benefit from acute repair
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bone work
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periarticular fractures may be fixed acutely or spanned with external fixator depending on surgeon preference
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instrumentation
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place knee-spanning external fixator in 20-30 degrees of flexion with knee reduced in AP and sagittal planes
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Early ligamentous reconstruction (<3 weeks)
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approach
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arthroscopic versus open
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arthroscopic may not be possible if large capsular injury and creates a risk of fluid extravasation and compartment syndrome
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PLC and PMC require open reconstruction given subcutaneous nature and proximity to neurovascular structures
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soft tissue work
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arthroscopic reconstruction of ACL and/or PCL
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address intraarticular pathology (menisci, cartilage defects, capsular injury)
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open repair versus reconstruction of collateral ligaments
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- outcomes
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recent systematic review suggests that patients who undergo staged reconstruction have a higher likelihood of having good to excellent outcomes
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acute (< 3 weeks) reconstruction is associated with a higher incidence of residual instability and stiffness that is resistant to nonoperative interventions
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COMPLICATIONS
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Vascular compromise
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incidence
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5-15% in all dislocations
- 40-50% in anterior or posterior dislocations
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risk factors
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KD IV injuries have the highest rate of vascular injuries
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treatment
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emergent vascular repair and prophylactic fasciotomies
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Stiffness (arthrofibrosis)
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incidence
- most common complication (38%)
- most common complication (38%)
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risk factors
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more common with delayed mobilization
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treatment
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avoid stiffness with early motion
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arthroscopic lysis of adhesion
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manipulation under anesthesia
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Laxity and instability
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incidence
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37% of some instability, however, redislocation is uncommon
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treatment
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bracing
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revision reconstruction
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Peroneal nerve injury
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incidence
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25% occurrence of a peroneal nerve injury
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50% recover partially
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risk factors
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male gender
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increased BMI
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associated fibular head fracture
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treatment
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AFO to prevent equinus contracture
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neurolysis or exploration at the time of reconstruction
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nerve repair or reconstruction or tendon transfers if chronic nerve palsy persists
- dynamic tendon transfer involves transferring the posterior tibial tendon (PTT) to the foot
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PROGNOSIS
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Complications frequent and rarely does knee return to a pre-injury state
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